Clinical Approach

How to Approach a Patient with Acute Kidney Injury

Acute kidney injury complicates up to 20% of hospital admissions and is independently associated with increased mortality, prolonged hospitalization, and long-term CKD progression. The approach centers on rapid categorization into prerenal, intrinsic renal, or postrenal causes — each with distinct management.

⚠️ Must-Not-Miss Diagnoses

Obstructive uropathy (treatable with catheter or stent), rapidly progressive GN (requires urgent kidney biopsy and immunosuppression), renal artery occlusion, acute cortical necrosis.

Differential Diagnosis by Key Features

Clinical FeatureThink OfPriority
Volume depletion: vomiting, diarrhea, bleeding, poor intakePrerenal AKICommon — fluids first
NSAIDs, ACE inhibitors, contrast, aminoglycosides, vancomycinNephrotoxic ATNCommon — stop offending agent
Inability to void, suprapubic fullness, BPH, pelvic malignancyObstructive uropathyHIGH — Foley catheter immediately
Hematuria + RBC casts + proteinuria + hypertensionGlomerulonephritisHIGH — nephrology urgently
Recent contrast or NSAID, no casts, fractional excretion lowContrast nephropathy / prerenalModerate
Muddy brown granular casts, prior hypotension or nephrotoxinATNModerate
Rash + eosinophilia + recent new medicationAcute interstitial nephritisModerate — stop drug

Systematic Approach

Exclude Obstruction

Bladder ultrasound immediately

Postrenal AKI is the most immediately reversible cause. Bedside bladder ultrasound takes 60 seconds and rules out urinary retention. Renal ultrasound evaluates for hydronephrosis from upper tract obstruction.

Assess Volume

Clinical exam + response to fluids

Examine for signs of hypovolemia (dry mucous membranes, poor skin turgor, orthostatic hypotension) vs euvolemia vs hypervolemia. Judicious fluid challenge helps distinguish prerenal from intrinsic in unclear cases.

Urine Studies

FENa, urine microscopy, urine protein

FENa <1%: prerenal (kidneys retaining sodium). FENa >2%: ATN (tubules can't retain sodium). Exception: contrast nephropathy and early obstruction may show low FENa. Granular muddy brown casts = ATN. RBC casts = GN.

Medication Review

Remove nephrotoxins immediately

Review all medications: NSAIDs, ACE inhibitors/ARBs (reduce GFR in low-flow states), aminoglycosides, vancomycin, contrast agents, calcineurin inhibitors. Stopping the offending agent is often the most important intervention.

Practice This Clinical Approach on ReasonDx

Work through simulated patient cases with AI coaching that guides your reasoning step by step.

Start Free →