Clinical Approach

How to Approach a Patient with Syncope

Syncope affects up to 40% of the population at least once in their lifetime and accounts for 1-3% of emergency department visits. The challenge is that most syncope is benign — but a minority has a life-threatening cardiac cause that is entirely treatable if identified.

⚠️ Must-Not-Miss Diagnoses

Ventricular tachycardia/fibrillation, complete heart block, STEMI, aortic stenosis, HCM, PE, aortic dissection, subarachnoid hemorrhage.

Differential Diagnosis by Key Features

Clinical FeatureThink OfPriority
Exertional syncope, family history of sudden death, young patientHypertrophic cardiomyopathy / structuralHIGH
Palpitations before syncope, rapid onset/offsetArrhythmiaHIGH
Prodrome (nausea, diaphoresis, vision graying), prolonged standing, warm environmentVasovagalLow
Position change (lying → standing), antihypertensives, elderlyOrthostatic hypotensionLow-moderate
After coughing, micturition, defecationSituational syncopeLow
Focal neuro deficit, headache before syncopeNeurologic cause (SAH, seizure)Moderate

Systematic Approach

History

Prodrome, circumstances, witnesses

The history is your most powerful diagnostic tool. Was there a prodrome (vasovagal) or no warning (arrhythmia)? What was the patient doing? What did witnesses observe? How quickly did they recover?

ECG

Obtain in every patient with syncope

ECG is mandatory. Look for: prolonged QT (Torsades risk), pre-excitation (WPW), Brugada pattern, heart block, ischemic changes, RV strain (PE).

Risk Stratify

ROSE or San Francisco Syncope Rule

High-risk features requiring admission: abnormal ECG, signs of heart failure, hematocrit <30, shortness of breath, SBP <90. Low-risk young patients with classic vasovagal story can often be discharged.

Orthostatics

Check in all patients

Measure BP lying and standing. Drop >20 mmHg systolic or >10 mmHg diastolic with symptoms = orthostatic hypotension. Review medications — antihypertensives and diuretics are common culprits.

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